Unfortunately, unconsciousness (sleep and coma) is also produced by the workings of neurons in a human brain.
That really only provides a location. SRIP provides one type of action that is probably necessary for human consciousness; and it is the particular types of actions, the architecture that we need to pin down. I think most of us agree on the location of it.
Wasp, sequel to your detailed reply to Clive on pain complexity (
post #3977), just focussing on "pain", do you think it's possible to state necessary and/or sufficient conditions for consciousness of pain, in terms of neurons? Is there a minimum level of electrochemical activity before a pattern of neurons firing is conscious for the subject? Does it ever make sense to speak of unconscious "pain" (neurons firing along dedicated pain pathways which the subject isn't conscious of; or perhaps anesthetized pain, where the enzymes that cause the pain neurons to fire are inhibited by drugs)? Beyond just pain, are there certain subsystems of the nervous system which must be involved for the subject to be conscious of the neurons firing (i.e., is activity in certain regions always unconscious; always conscious)?
Maybe a pointless sidetrack, but I'm curious how much "consciousness" can be pinned down to certain regions of the brain / nervous system; certain threshholds of nervous activity; perhaps even certain patterns of enzyme catalysis or neurons firing? It's well known of course that certain frequencies of neurons firing as measured by EEGs are indicative of certain types of consciousness -- (Beta [12-30 Hz] and active concentration, for example) -- and certain regions more active than others in these states: can we be any more specific (layman deferring to your expertise; you'd hinted at an answer in your earlier reply re the five pain pathways)?
I was fascinated by your discussion of pain asymbolia, where patients can feel pain yet not suffer, not be repelled or motivated by feeling it. Naively, I would have expected the pain 'quale'
was the motivation for acting to alleviate the pain. Yet it appears the consciousness of the pain, the 'quale', can be separated from the motivation to act on it. Any complementary cases you know of, where patients react to 'painful' stimuli that they don't 'feel' ("painful" here would translate to what would cause pain absent the condition: not sure what to call it... "pain pzombia"?)?
Whew, that's a lot of questions... sorry.

(& a slightly premature happy new year!)
ETA: whoops... looks like you've answered most of my last question in a subsequent reply to Orbini (
post #3978). *sigh* just ignore me...
