ServiceSoon
Graduate Poster
- Joined
- Oct 20, 2007
- Messages
- 1,745
I accept your insinuation for why H1N1 may have been more deadly for the young. It appears the opposite is true for COVID-19; this disease is more deadly for the biologically old. Would you be interested in hazarding a guess on why COVID-19 has this opposite effect?Furthermore nearly all flue strains are relatively closely related to existing strains that have circulated before, so a lot of people have at least some resistance which slows the spread of the virus. It’s been hypothesized that the reason why H1N1 was abnormally hard on younger people is that people over 35 had already been exposed to a related strain.
The rapid spread of COVID 19 could indicate that there is no such protection built into the population.
When considering the worlds extreme measures taken in response for COVID-19 and contrasted with our lack of concern for H1N1, it invokes thoughts for the existence of ageism. I'm very curious what the models predicted when H1N1 was first discovered.
I keep reading claims that immunity to COVID-19 does not exist and use that statement to justify our hysteria. If an estimated 80% of those infected by COVID-19 are asymptomatic, isn't that an indication that they have immunity? In that cohort of 80%, SARS-CoV-2 does not elicit a pathological disease response. That is immunity if I understand the term correctly.