It depends on what kind of memory it is, the circumstances surrounding the event, and the trigger for recalling the memory. What you posted doesn't apply wholesale to every situation, reconsolidation isn't a given.
http://www.human-memory.net/processes_encoding.html
"Human memory is fundamentally associative, meaning that a new piece of information is remembered better if it can be associated with previously acquired knowledge that is already firmly anchored in memory. The more personally meaningful the association, the more effective the encoding and consolidation. Elaborate processing that emphasizes meaning and associations that are familiar tends to leads to improved recall."
"
Many studies have shown that the most vivid autobiographical memories tend to be of emotional events, which are likely to be recalled more often and with more clarity and detail than neutral events. One theory suggests that high levels of emotional arousal lead to attention narrowing, where the range of sensitive cues from the stimulus and its environment is decreased, so that information central to the source of the emotional arousal is strongly encoded while peripheral details are not (e.g. the so-called “weapon focus effect”, in which witnesses to a crime tend to remember the gun or knife in great detail, but not other more peripheral details such as the perpetrator’s clothing or vehicle)."
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2265099/
"The potential for altering emotional memories during or immediately after retrieval is an intriguing possibility that has been explored extensively in animal models.
The alteration of postretrieval emotional memory traces in humans, however, has not been conclusively demonstrated. The “first wave” of reconsolidation research led to conflicting reports as to whether electroconvulsive therapy produced amnesia for reactivated memory traces in humans . Although more recent human research has suggested that the memory trace for a motor sequence task can be disrupted through a reconsolidation-type mechanism ,
the learning of a motor sequence is quite distinct from emotional declarative memories. Although animal work has provided several demonstrations of reconsolidation of hippocampally mediated memories, it is not clear to what extent the same reconsolidation mechanisms affect human declarative memory traces, especially for emotional events. The similarities in brain mechanisms that underlie rodent and human emotional learning situations, such as fear conditioning, extinction, and reward learning, suggest that the labile state that is thought to be affected in animal reconsolidation paradigms should also be manipulable during memory retrieval in humans.
However, evidence for reconsolidation of human emotional memory has thus far remained elusive."
http://www.ncbi.nlm.nih.gov/pubmed/26238574
"Memory reconsolidation is considered to be the process whereby stored memories become labile on recall, allowing updating. Blocking the restabilization of a memory during reconsolidation is held to result in a permanent amnesia. The targeted knockdown of either Zif268 or Arc levels in the brain, and inhibition of protein synthesis, after a brief recall results in a non-recoverable retrograde amnesia, known as reconsolidation blockade. These experimental manipulations are seen as key proof for the existence of reconsolidation. However, here we demonstrate that despite disrupting the molecular correlates of reconsolidation in the hippocampus, rodents are still able to recover contextual memories.
Our results challenge the view that reconsolidation is a separate memory process and instead suggest that the molecular events activated initially at recall act to constrain premature extinction."
What you guys are basing your position on are from these kinds of studies that are based on learning and the treatment for disorders that affect memory. Without consolidation/reconsolidation we would never be able to update anything we've ever learned. That kind of memory is distinctly different from a memory that results from an emotional event.
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3650827/
Three studies are of direct relevance to the hypothesis that reconsolidation mediates memory updating . Firstly, in human episodic memories, interference congruent with retrieval of a prior memory results in an incorrectly updated memory for a list of items . This finding is consistent with, though not directly demonstrative of, a role of reconsolidation in updating memories.
Moreover, a prior study of inhibitory avoidance learning in rats did not provide evidence that reconsolidation is functionally involved in linking new information to a reactivated memory. Using the doubly dissociable mechanisms of inhibitory avoidance memory consolidation and reconsolidation, Tronel et al. showed that second-order conditioning recruited consolidation processes selectively .However, linking new information to an old memory can be viewed simply as new learning based upon evoked memories, which would be expected to necessitate consolidation mechanisms, rather than true memory updating."
"
Therefore, memory reconsolidation may well prove to be the mechanism by which memories are updated through further experience, though it remains to be determined whether reconsolidation plays a similar functional role in other forms of memory updating, such as memory weakening or changes in memory content."
"Memory reconsolidation is a rapidly expanding field of research, which is becoming widely accepted as a fundamental process in long-term memory. While advances are continually being made in terms of the pharmacological and cellular mechanisms of reconsolidation, these do not address the fundamental question of the role of memory reconsolidation in memory persistence. In this review, I have proposed that the function of memory reconsolidation may be to mediate the updating of a memory in order to maintain its adaptive relevance. This memory updating hypothesis of reconsolidation has the potential to account for when reconsolidation does and does not take place. Indeed, I suggest both that reconsolidation is in fact a universal property of memories and that it is engaged specifically under conditions of memory updating.
A major question that emerges is that of how memory reactivation is determined at the mechanistic level depending on whether a memory should be updated."
Further reading suggests that we'll have to rethink all of the above because they have discovered protein prions that play a role in the different kinds of memory processes.
http://newsroom.cumc.columbia.edu/blog/2015/07/02/long-term-memories-and-prions/
"When long-term memories are created in the brain, new connections are made between neurons to store the memory. But those physical connections must be maintained for a memory to persist, or else they will disintegrate and the memory will disappear within days. Many researchers have searched for molecules that maintain long-term memory, but their identity has remained elusive."
"As long as these aggregates are present, Kandel says, long-term memories persist. Prion aggregates renew themselves by continually recruiting newly made soluble prions into the aggregates. “This ongoing maintenance is crucial,” said Dr. Kandel. “It’s how you remember, for example, your first love for the rest of your life.”
Well I guess that explains why statin drugs can sometimes affect memory. Without cholesterol, protein synthesis stops in the brain.