Mouthfire said:
I agree with a lot of what you've said. I'm sure that as a teacher, you have a lot of experience with these issues. I will disagree with a few points, however, as recent published research have given us new insights into ADHD.
Thanks for the input, Mouthfire.
I have to point out that my background is a mix of microbiology and biochemistry pathology and teaching. Not a lot of psych' in either of those, although in teaching I've needed to learn a lot of different areas to do with behavioural and cognitive development. I still cannot confess to being anything of an expert in the field.
Personally, I would have problems lumping ADHD together with Pervasive Developmental Disorders (or Autism Spectrum Disorders), such as Autism and Asperger's. I know that some professionals do, but I prefer to reserve the PDD designation to entities that have a prominent developmental delay component. Although patients with ADHD can have developmental delay, I find that most ADHD children have normal psychomotor development, despite their learning difficulties.
I should have been clearer in expressing this viewpoint, as it's not I one I share wholeheartedly either. I must admit, I hesitate labelling a lot of conditions and withold a lot of opinions to do with it. Having a much more traditional background when it comes to communicable and biochemical pathology, I don't feel all that brave in arguing psychopathology from a psychological point of view. Label it what you want; I'm more interested in accomodating the repurcussions.
Recent studies, however, do suggest that there is a true pathological basis of ADHD, and currently, I would be against classifying ADHD a trait, rather than as a disease. Please see below for further elaboration.
Pathology is typically described less as a variation of the norm and more as a situation described as the typical variants being perverted by an environmental change. If ASD (or, insert term here) is found to be caused largely by an environmental influence, I'll agree that it can be described as being more pathological rather than genetically variant. Again, the only thing I know is that we don't have anything conclusive yet, but we do have a whole of lot people looking.
I'm sure I'll have a different opinion in years to come.
That is true. However, as I stated before, ADHD is not really related to Asperger's. In fact, one of the requirements for diagnosing ADHD is that they have difficulties in multiple environments, not just at school. If a child were having attention problems related to only school, I would suspect either specific learning disorders or social problems before ADHD.
ADHD and Asperger's are indeed unrelated in terms of definitive behaviours. But using the school environment exclusively to diagnose anything is always troublesome.
The problem I keep coming back to (and, it seems, the same problem you see) is that we try to take a range of traits and enclose them neatly into an encyclopedic article. We love having tables which we can insert names and numbers into, and we'll look for any way to do that. As I said before, our traditional view of treating abnormalities with a view of cause and effect (seen in the numerous newspaper articles we come across yearly which are headed
'Scientists Discover the Gene For...') can mislead us.
I agree wholeheartedly. Diagnosing ADHD from normal behavior is very difficult. Furthermore, most psychologists, psychiatrists, and neurologists do not have enough time to properly diagnose it, as that would involve mutiple evaluations lasting hours at the least.
From my personal experience, I feel that a lot of diagnosic tests have the same feeling as a personality test, which further prompts me to see some of these conditions as being variant traits. In addition, sometimes the social background of people can further impact (note, I'm not saying
causes) the severity of the behaviour. In Australia, ADHD is diagnosed in a lot of Indigenous children, with the extrapolation that it is a genetic thing. However it seems to be more prominant in children raised in Aboriginal communities rather than in urban settings. I can't find any papers discussing the reasons for this, but then I haven't looked that hard.
It would be interesting to investigate.
Recently, there have been multiple studies delineating what exactly is different in ADHD patients. Many studies, in particular one from Mostofsky et al (2002), found that the total cerebral volume is reduced in ADHD children (compared to control). In particular, the frontal lobes are reduced, which control behavior and self-regulation. Castellanos et al (2002) also found that the right hemisphere is smaller in ADHD children. The right hemisphere is involved in regulating attention, which is notably awry in ADHD.
I read an abstract for the Mostofsky paper, and found it interesting. But it doesn't say a whole lot, other than suggesting there are physiological signs of the condition that may assist diagnosis. However, that said, I would be shocked if there was not physiological variations in people who expressed behaviours such as those defining ASD conditions.
This is why most neurological and psychiatric professionals have problems classifying ADHD as normal. There is simply too much evidence that there is a biological basis. Furthermore, there has been evidence that some of the pathophysiology behind ADHD is from deficient neurotransmitters. In particular, dopamine and norepinephrine have been implicated. Dopamine is involved in working memory, learning, and emotion. Norepinephrine is involved in maintaining attention and alertness.
Why can't this again be a variation? You've made the mistake of assuming 'normal' is a sub-group of the population, and ADHD sufferers are outside of this. I've already confessed to not being an authority on the topic, and am happy to be wrong, but considering what I
have read on the topic, and experienced personally, I still think we get carried away with observing a desire for definitive lines.
Maybe we should ask, 'At what concentration does neurotransmitter X have to be reduced to for the person to have Condition Y?' Looked at from this perspective, it's easier to see that we all exist on a scale of behaviours.
As for food additive, allergens, etc... I personally have not seen a lot of evidence for this.
Yeah, I don't have a strong opinion on this either way. A lot of anecdotal evidence, mind you, but I'd like to see some strong evidence from a good study before I start to argue it.
A final point regarding Ritalin and other psychostimulant medications: The theory behind the use of these meds is not for the stimulant or metabolic effect, per se. Ritalin et al increase the amount of dopamine and norepinephrine in the central nervous system. A new medication, in fact, (Strattera) has no stimulant effect, but increases dopamine and norepinephrine levels. It has shown early positive results after being placed on the market, and I have professionally had some good results with it.
When I read this, I realised I did make a mistake. You're 100% right and I do remember reading this, and stand corrected.
Thanks for the response, Mouthfire.
Athon