Is sleep paralysis really an explanation?

What in the world is a "woo hijacking"???

It is when someone starts a legitimate topic until someone comes along with a completely off-the-wall post that forever changes the direction of the thread, thus dooming the OP from getting much more consideration.
 
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This physical loss can range from molecular loss, such as the loss of insulin or glucose control in diabetics to an amputee as well as those who are incapable of normal physical means of protecting themselves, such as those who are paralyzed, in a coma, ill, etc.
Or just a low blood sugar since you haven't eaten since dinner and it's 4 am?
 
To note, before getting into explanations…at present time I am in college to learn how to find the best way to test what I’ve figured out through my own experiences of SP. Other things involved include family medical history and the basics of knowledge in the life sciences. I am all on the side for science and the scientific method. Questions I am without answers for I will be honest and take those questions to heart. These are the questions I know I’ll need to find answers for to complete the missing pieces of information. Personally, I am my own case study and so is my grandmother who passed away in 1999 from complications due to the ignorance in the medical field on conditions like cataplexy and catalepsy, which she had as well as sleep paralysis.

In addition to Arthwollipot's questions:
What leads you to consider these two factors?

The senses themselves are reliant on physical contact in any form. Why would it be any different for ESP activity considering ESP is a sensory activity? You must have the mechanics to output the sense and the mechanics, provided they work, rely on sensory stimuli, which requires physical contact with energy particles from the substance being sensed.
As of physical loss…so far those who I have spoken to who have a type of physical loss commonly complain of sleep disturbances that fit some or all of the definitions of SP or similar to.

It is already known that sensory perception does make up for a loss of another sense due to a physical limitation…maybe the mechanics are damaged…hormones disrupted…neural pathways damaged, etc. For example, those with poor eyesight are known to have a heightened sensed of hearing. Those who have a loss of hearing may have heightened senses of touch, which I suspect knowing that Helen Keller, who was both blind and deaf was capable of learning through the sense of touch, thus touch may be the a typical compensation for the loss of hearing. Now, this is sense compensating for another sense, but it is still the brain compensating for a physical loss.

Another thought of reasoning behind this is what I call normal ESP activity (for lack of a better term), which is largely seen in children prior to puberty. Taking children as for example, their experiences with ghosts and other odd ‘paranormal’ type occurrences are very common and it is also known the pineal gland is well engorged in children until puberty, when it shrinks.
Children are naturally vulnerable to danger without physical loss, but that is because of the lack of physical development simply due to age. They are still developing thus have a lack of an X degree of physical ability.

The pineal gland is a top candidate as being the organ responsible for the translations of sensory information between inner and outer environments, considering the pineal gland is stimulated by heat and light sources from both inner and outer environments, is smack in the center of the thalami halves, does secrete a hormone that has similar chemical structures as hallucinogens found in plants (I know the name as pinoline) and is found in the retina as an oxidative agent with or for vitamin E. I think pinoline acts as a chemical solution to develop a corresponding image to the neural impulses that pass through the eyes…whether awake or asleep. This would account for all types of vision and both sight and sound. Sight and sound are the most common forms of ESP.

Another thought to this aspect of physical loss…I’ll use Stephen Hawking as an example.

All neural outputs come from the same neural inputs. Some of this input is meant for cells in the body the brain transfers. Everything goes to the brain first, then to where ever in the body. Stephen Hawking’s lack of physical mobility means nearly all of the sensory inputs are restricted to his brain. Talk about extrasensory!! As much as I would love to have this man’s dreams, I would hate to have his nightmares.


What specifically leads you to believe that any of these may be linked?

Through similar behavior. If neural impulses can be diverted to the thalami meant for somewhere else, then any channels of communication could be disordered just the same. See above answer about Stephen Hawking.
As of the finer lines between life and death, this does take a twisted turn. This comes from family medical history of my grandmother who had narcolepsy, cataplexy, catalepsy, central and obstructive sleep apnea, and insomnia and sleep paralysis. She also had a long list of other metabolic problems with her thyroids and adrenals.

The connection I made is with the pineal gland. It is an organ that does control the rate the metabolic system flows and controls the sleep and wake states. All sleep disorders are directly related to the pineal gland and the pineal gland is directly related to the pace of the heartbeat, blood pressure and breathing, thus the flow rate of the metabolic system. Cataplexy and catalepsy have identical metabolic behaviors as hibernation, deep meditation and coma. The difference is that cataplexy and catalepsy are automatic and sudden. The difference between catalepsy and cataplexy is muscle tone. Cataplexy, muscle tone is relaxed, whereas catalepsy, muscle tone is rigid.
Metabolism is brought down to a crawl in each of these examples. Some to a slower crawl than others to the point of feigning death. My grandmother was pronounced dead three times in her life. Once she actually woke up in the morgue. I am currently trying to hunt down medical records. Not an easy thing to do…especially when one of those deaths occurred in 1924, shortly after she was born.


Which specific pathways would these be, and how do they account for claims of ESP activity?

I think some of the answers for this can be sorted out through things already written. I’m still a student and not familiar with all the proper terminology yet.

For that matter, how would impulses meant for tissues that are a part of the immune system manage to encode (assuming that they somehow found their way to V1) such a specific visual experience (alien) in the visual system?

As mentioned, all neural outputs come from the same neural inputs, meaning the outputs, regardless of expression, are made from the same chemical compositions. It is the mechanism that allows for the translation of these chemical compositions. If the impulse shoots off to the thalami and eyes and ears work…those chemical signatures would be translated into image and sound. If impulses are sent to tissues then those chemical signatures would be translated through mechanism of those tissues.

This sounds preposterous without evidence, I hope you realize that.

I know…lol. but I do my best to stick with what is already considered accepted scientific knowledge and I always knew better to keep this study based on biology and the sleep paralysis gave me something real and tangible to work with. Though studying sleep paralysis for 26 years, it wasn’t until 1994 when ESP was included. I use to be a big skeptic of ESP type things. I was just as highly critical as anyone else. It was considered entertainment. But once the first hallucination I had during SP came true, all that changed.
The hallucination was in true form, meaning, the objects in the hallucination were as they really were when the event actually happened, except for one significant detail, which was a direct result of remembering the hallucination at the proper time to prevent a much more damaging outcome. I got to act on my intuition. The most predominant features of this particular hallucination is that it happened nearly 3,000 miles away from where the hallucination took place…and several years later.


This makes no sense whatsoever. Word association doesn't work on the cellular level, I am afraid.

This is where you are wrong. I’m actually quite surprised to come across such a comment. Everything biological works on a cellular level. Everything cellular works on molecular structures. Molecular structures are composed of atoms. Everything is made of atoms, including the sounds, sights, touches, etc. of words.


I am looking forward to some actual evidence that supports your position. Have any?

If I had evidence, it’d be theory now wouldn’t it? Just kidding…though it would…as for a real answer…

The metabolic tie is direct with the pineal gland and it is known the pineal gland is a definite organ that controls the metabolic system.

The activities of ESP seem to be automatic behavior and automatic behavior is well accepted as instinctive behavior and instinctive behavior is well accepted to be a behavior shared in all organisms, such as the instinct to procreate and to fight or flee.

Also, personally, every hallucination I’ve had seems to relate to health and safety and it is something I’ve noticed the same with other people.
I have looked into other organs in other organisms that seem relative to our own pineal gland, further supporting some of the hypotheses. These organs are in direct relation to the organism’s metabolism and the means of translating information between inner and outer environments and stimulated by heat and light sources. These organs include the visible third eye on some reptiles, the Jacobson organ in reptiles without a visible third eye (most notably snakes), the optic lobe in fish and eyespots on worms and parasites.

A somewhat off in left field thought about the pineal gland…and if true, would also be true for each of these other organs…is that I think the pineal gland originates as the zygote. Strange yes, though I’ve had stranger thoughts.

I think you are quickly going to find that the motto here is: "Show me the evidence." Furthermore, stating what you believe without providing any reasons is worthless. My first two questions address that point. What led you to those conclusions? You hypothesize that ESP is metabolic in nature, but have nothing to point to back that up. Simply it ascribing to some conditions that can be much more easily and convincingly explained by other means just doesn't cut it.


As stated, there’s so much more information, that I left it open for these types of questions. I’ve got 26 years worth of SP experiences to help me out. As of hardcore physical evidence, well, I’m working on that. That’s what I’m hoping college helps me out with…learning a way to test this out and obtain the evidence in a manner to the scientific method.

There is one situation, which includes a series of SP, I might have enough proof along with existing information about other sources, to nab at least an interest in key areas of the biological sciences for further investigation. Nothing to necessarily prove the existence of ESP, but a means to have key people in the sciences to at least consider some support for further research. Dates are borderline, but verifiable. This includes perceptions during SP about my grandmother when there were problems with her health and the doctors were all at a loss of what was wrong and how to help her. I figured it out and at the time I was a high school dropout and an unemployed housewife nearly three thousand miles away with the aid of the hallucinations produced during SP.

What I did was write my grandmother a letter. She was incapable of reading it herself. My uncle read it for her and placed it under her pillow. I had a letter she wrote to me some time before and I kept it under my pillow. Every time I laid down to sleep, I would wake up in SP and have a barrage of hallucinations and they showed me things like explosions, gun powder, things automatic and change shown in the form of metal coins. Gases, x-rays, the colon and intestines. Also a view from a single eye…that was neat…oxygenation was also in several hallucinations. Each of these and other things kept showing up. Even my grandmother with her amputated leg from cellulitis showed up once in a while.

I busted out with all sorts of resources…dictionaries and textbooks mostly and looked for thing that are common between these hallucinations and my grandmother’s condition. I did keep in touch with family about medications, changes in her responses, etc…looking for common things in that. I had made a list of things I felt the doctors should check and emailed it to my mother who told them. According to her, they all agreed, but had to wait for her seizures to stop, but she had died before that happened. Though I do wonder if she had actually died on the autopsy table. I don’t know, but considering she’s been mistaken for dead more than once in her life, it is a possibility.

The autopsy was done shortly after her death, but the report was released at the end of July 1999. She died in May of that year. I jotted down those things I felt the doctors should check along with brief descriptions of the hallucinations…such as what I’ve described here and mailed it to myself before the autopsy report was available. The postmark is dated the beginning of July. This is what I mean by dates being borderline, but verifiable or should be anyway. It is a documented death. I was prohibited from communicating with her attending physicians, because hospital policy requires patient information be given in person and I was nearly 3,000 miles away. I did speak with her sleep doctor, though only asked him if her medication to control the cataplexy was based on the pineal gland hormone, pinoline. He said yes. Now, even with this, there are limitations that kept me from gaining knowledge from him about her present condition. Her sleep doctor is also under policy of patient confidentiality and was restricted from interfering with her care at the hospital she was at, because he was not on their list of participating doctors. He belonged to another hospital.

The microscopic descriptions of her autopsy report seems to reflect a lot of what I was picking up in the hallucinations…oxidation, autolytic changes, pineal calcification I also noted interestingly. The explosions and gun powder and gases are from metabolic upheaval when she was given meds like lasix…the phosphorus connection (or is that potassium?….I always get the two mixed up in which one is used as an explosive, but also used in denture care products. Having her medical lab reports would be very interesting.

I really don’t know just how much this would hold as any ounce of proof of having perceptions about another from a long distance, at the time, with much less knowledge than what I have now about biology and medicine and with restrictions of knowledge from professionals in the field in her direct care.

The envelope remains sealed.

Pardon any typos...this is really long to comb through...
 
It is when someone starts a legitimate topic until someone comes along with a completely off-the-wall post that forever changes the direction of the thread, thus dooming the OP from getting much more consideration.

I don't mean to take any attention away from anyone. I did reply to a post on topic. If appropriate...perhaps someone could move what I've apparently started to its own thread?

Just a thought. :)
 
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Or just a low blood sugar since you haven't eaten since dinner and it's 4 am?

Good question. Something to consider looking into more. Though experiences, if any, I'd think would reflect a mild termporary disturbance considering there is a temporary imbalance, so long as the imbalance is tended to equate metabolism. Sugar maintenence is very important to metabolism. Any disruption could bring on ESP activity if perhaps the link to metabolism can hold true.
 
To note, before getting into explanations…at present time I am in college to learn how to find the best way to test what I’ve figured out through my own experiences of SP. Other things involved include family medical history and the basics of knowledge in the life sciences. I am all on the side for science and the scientific method. Questions I am without answers for I will be honest and take those questions to heart. These are the questions I know I’ll need to find answers for to complete the missing pieces of information. Personally, I am my own case study and so is my grandmother who passed away in 1999 from complications due to the ignorance in the medical field on conditions like cataplexy and catalepsy, which she had as well as sleep paralysis.



As stated, there’s so much more information, that I left it open for these types of questions. I’ve got 26 years worth of SP experiences to help me out. As of hardcore physical evidence, well, I’m working on that. That’s what I’m hoping college helps me out with…learning a way to test this out and obtain the evidence in a manner to the scientific method.

There is one situation, which includes a series of SP, I might have enough proof along with existing information about other sources, to nab at least an interest in key areas of the biological sciences for further investigation. Nothing to necessarily prove the existence of ESP, but a means to have key people in the sciences to at least consider some support for further research. Dates are borderline, but verifiable. This includes perceptions during SP about my grandmother when there were problems with her health and the doctors were all at a loss of what was wrong and how to help her. I figured it out and at the time I was a high school dropout and an unemployed housewife nearly three thousand miles away with the aid of the hallucinations produced during SP.

What I did was write my grandmother a letter. She was incapable of reading it herself. My uncle read it for her and placed it under her pillow. I had a letter she wrote to me some time before and I kept it under my pillow. Every time I laid down to sleep, I would wake up in SP and have a barrage of hallucinations and they showed me things like explosions, gun powder, things automatic and change shown in the form of metal coins. Gases, x-rays, the colon and intestines. Also a view from a single eye…that was neat…oxygenation was also in several hallucinations. Each of these and other things kept showing up. Even my grandmother with her amputated leg from cellulitis showed up once in a while.

I busted out with all sorts of resources…dictionaries and textbooks mostly and looked for thing that are common between these hallucinations and my grandmother’s condition. I did keep in touch with family about medications, changes in her responses, etc…looking for common things in that. I had made a list of things I felt the doctors should check and emailed it to my mother who told them. According to her, they all agreed, but had to wait for her seizures to stop, but she had died before that happened. Though I do wonder if she had actually died on the autopsy table. I don’t know, but considering she’s been mistaken for dead more than once in her life, it is a possibility.

The autopsy was done shortly after her death, but the report was released at the end of July 1999. She died in May of that year. I jotted down those things I felt the doctors should check along with brief descriptions of the hallucinations…such as what I’ve described here and mailed it to myself before the autopsy report was available. The postmark is dated the beginning of July. This is what I mean by dates being borderline, but verifiable or should be anyway. It is a documented death. I was prohibited from communicating with her attending physicians, because hospital policy requires patient information be given in person and I was nearly 3,000 miles away. I did speak with her sleep doctor, though only asked him if her medication to control the cataplexy was based on the pineal gland hormone, pinoline. He said yes. Now, even with this, there are limitations that kept me from gaining knowledge from him about her present condition. Her sleep doctor is also under policy of patient confidentiality and was restricted from interfering with her care at the hospital she was at, because he was not on their list of participating doctors. He belonged to another hospital.

The microscopic descriptions of her autopsy report seems to reflect a lot of what I was picking up in the hallucinations…oxidation, autolytic changes, pineal calcification I also noted interestingly. The explosions and gun powder and gases are from metabolic upheaval when she was given meds like lasix…the phosphorus connection (or is that potassium?….I always get the two mixed up in which one is used as an explosive, but also used in denture care products. Having her medical lab reports would be very interesting.

I really don’t know just how much this would hold as any ounce of proof of having perceptions about another from a long distance, at the time, with much less knowledge than what I have now about biology and medicine and with restrictions of knowledge from professionals in the field in her direct care.

The envelope remains sealed.

Pardon any typos...this is really long to comb through...


I'd suggest starting your own thread in General Skepticism and The Paranormal. "My experiences with ESP and my Grandmother" or something.

There's also a thread about DMT and near-death experiences that gets into speculation about the pineal gland a little--it's in Science, Mathematics, Medicine, and Technology, I think.

You might also start some thread about afterlife and the soul, etc. in Religion and Philosophy, or add to one of those.
 
Hey cj.23 :) Hello...LOL. Didn't mean to rob a thread, but it is originally a reply to you. I'd like to stay here if it's OK, considering this is the topic at hand.

Thanks.
 
I'd suggest starting your own thread in General Skepticism and The Paranormal. "My experiences with ESP and my Grandmother" or something.

There's also a thread about DMT and near-death experiences that gets into speculation about the pineal gland a little--it's in Science, Mathematics, Medicine, and Technology, I think.

You might also start some thread about afterlife and the soul, etc. in Religion and Philosophy, or add to one of those.

My experiences are fairly solid on sleep paralysis. This is a very appropriate topic. But thanks for the references. I'll check them out :)
 
I was just thinking about this. Is "sleep paralysis" actually meaningful as an explanatory hypothesis? We all know what it is I'm guessing, and most of us would classify certain experiences as it, but as we lack any actual empirically testable hypothesis as far as I know, or even probable physiological mechanism with supporting evidence does it actually mean anything? It's a hypothesis, not a theory?

Dunno. Thought I'd ask

cj x

Huh?

Sleep paralysis has been studied quite a bit in sleep labs. It's related to REM atonia.
 
I was just thinking about this. Is "sleep paralysis" actually meaningful as an explanatory hypothesis? We all know what it is I'm guessing, and most of us would classify certain experiences as it, but as we lack any actual empirically testable hypothesis as far as I know, or even probable physiological mechanism with supporting evidence does it actually mean anything? It's a hypothesis, not a theory?

Dunno. Thought I'd ask

cj x

My experiences are fairly solid on sleep paralysis. This is a very appropriate topic. But thanks for the references. I'll check them out :)


I see that Miss Whiplash has beaten me to it, but I'll post the OP again for emphasis.

The OP is asking whether sleep paralysis explains things like reports of UFO abductions, ghost sightings, etc. How much do we really know about it?

Answer, quite alot, both from the subjective and the objective perspective.

Sleep paralysis and ESP are usually separate subjects, so if you want to combine them (and get more responses) start your own thread.
 
Having experienced sleep paralysis several times in my life. I have to say that you'd have to be crazy if you think aliens were responsible. Also, I've been able to break out of this paralysis several times and I tell yah, there were no little green men standing by my bed.
 
I see that Miss Whiplash has beaten me to it, but I'll post the OP again for emphasis.

The OP is asking whether sleep paralysis explains things like reports of UFO abductions, ghost sightings, etc. How much do we really know about it?

Answer, quite alot, both from the subjective and the objective perspective.

Sleep paralysis and ESP are usually separate subjects, so if you want to combine them (and get more responses) start your own thread.

And my reply responded to the OP's question. Am I supposed to ignore people who happen to ask about what I replied?
 
No, you're not, but when that starts to become a topic on its own, then it's a good idea to take it to a new thread, and save the mod team from intervening to split it out.

I'd advise that's the best course of action, and future posts in this thread could keep to the topic as posed in the OP.
 
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Huh?

Sleep paralysis has been studied quite a bit in sleep labs. It's related to REM atonia.

As far as i know REM atonia is just the medical name for sleep paralysis. Yes we have a vague idea that the body paralyses in REM sleep, and that the even some ideas as to how - but no actual description of the mechanism. That is what interests me.

Someone says "I had this weird experience". I say "oh that s sleep paralysis". They say "cool, whats that..." So I respond "it's REM atonia persisting after you wake up." Person "oh cool, er, how does that work". CJ "er, the neurological mechanism is not known or currently demonstrable. We have a few ideas, but no accurate description of the mechanisms involved." Person - "oh so it's psychobabble woo?"

Hence my concern. When we say these experiments are sleep paralysis, we are really just answering with a set of symptoms, and a tentative hypothesis - but we sound dead medical and scientific so people don't normally question.

Obviously I don't think sleep paralysis is aliens, ghosts or witches! I'm just saying that honestly we don't acualy really understand it yet.

cj x
 
As far as i know REM atonia is just the medical name for sleep paralysis. Yes we have a vague idea that the body paralyses in REM sleep, and that the even some ideas as to how - but no actual description of the mechanism. That is what interests me.

Someone says "I had this weird experience". I say "oh that s sleep paralysis". They say "cool, whats that..." So I respond "it's REM atonia persisting after you wake up." Person "oh cool, er, how does that work". CJ "er, the neurological mechanism is not known or currently demonstrable. We have a few ideas, but no accurate description of the mechanisms involved." Person - "oh so it's psychobabble woo?"

Hence my concern. When we say these experiments are sleep paralysis, we are really just answering with a set of symptoms, and a tentative hypothesis - but we sound dead medical and scientific so people don't normally question.

Obviously I don't think sleep paralysis is aliens, ghosts or witches! I'm just saying that honestly we don't acualy really understand it yet.

cj x

But that's all irrelevant to the question you actually asked, which was if sleep paralysis is really an explanation. The answer is yes it is. Sure, we don't know exactly how and why it happens, but that doesn't matter. We know that brain does something to cause the symptoms of sleep paralysis and that these include various things that can make people think they have had some kind of paranormal experience.

It's rather like saying that since we don't know exactly how a particular antibiotic works, we can't say that germ theory is a good explanation for disease. Sure, we don't know everything about how all diseases and all drugs work, but we still know that germ theory is the correct explanation.
 
We don't have perfect explanations, but we do have some.

First, REM atonia refers not to the sensation of sleep paralysis (which is something experienced during the waking state) but to the atonia that is evident during REM sleep and which is well documented by EMG studies during sleep (though, of course, REM atonia can refer to both situations -- the "normal" atonia of REM and the atonia of sleep paralysis).

From what I recall there are groups of lateral pontine and medial medullary reticular neurons that fire at high rates during REM sleep but are not active during NREM sleep, so they are thought to be important in generating the REM state. These same cells are silent during the waking state, but some become active when the head is lowered or with postural changes that involve reductions in tone -- so they may be important in generating the atonia of REM sleep or part of the relay.

From what we can tell, there is not really one particular area responsible for suppression of tone -- it seems to be distributed to several areas in the brainstem. Many of the cells responsible are probably cholinergic, since loss of muscle tone can be caused by injection of cholinergic agonists into the pons. The nucleus reticularis pontis oralis appears to be one of the major players since lesioning this area results in REM sleep without atonia (in humans this can be seen as REM behavior disorder).

I'd have to look up more details to be more precise.
 
But that's all irrelevant to the question you actually asked, which was if sleep paralysis is really an explanation. The answer is yes it is. Sure, we don't know exactly how and why it happens, but that doesn't matter. We know that brain does something to cause the symptoms of sleep paralysis and that these include various things that can make people think they have had some kind of paranormal experience.

Well one would have thought the brain was involved in any experience of a human, no matter if "paranormal" or not. So really it doe snot get us very far does it? Sure we understand what atonia is - just not how it works. We have some ideas, but nothing solid yet. So the even more confusing issue of why occasionally atonia might persist in to consciousness after the cessation of REM sleep - let's face it, it's still a mystery!

It's rather like saying that since we don't know exactly how a particular antibiotic works, we can't say that germ theory is a good explanation for disease.

No, germ theory is quite independent of the antibiotics efficacy. The latter can be demonstrated by medical trails and statistics, even if we are unsure of the physiological aspects. Even then we can't be sure that the antibiotic action and illness ending are definitely linked - correlation is not causality, and other factors may be involved. A good medical statistician ca make an overwhelming case though.

Sure, we don't know everything about how all diseases and all drugs work, but we still know that germ theory is the correct explanation.

It's not for all disease though is it? Prion disorders spring to mind immediately - cancer - etc, etc. And we might understand the principle, I think we do, but we still really should admit that we have very little evidence for any underlying model of the causality

cj x
 
We don't have perfect explanations, but we do have some.

First, REM atonia refers not to the sensation of sleep paralysis (which is something experienced during the waking state) but to the atonia that is evident during REM sleep and which is well documented by EMG studies during sleep (though, of course, REM atonia can refer to both situations -- the "normal" atonia of REM and the atonia of sleep paralysis).

From what I recall there are groups of lateral pontine and medial medullary reticular neurons that fire at high rates during REM sleep but are not active during NREM sleep, so they are thought to be important in generating the REM state. These same cells are silent during the waking state, but some become active when the head is lowered or with postural changes that involve reductions in tone -- so they may be important in generating the atonia of REM sleep or part of the relay.

From what we can tell, there is not really one particular area responsible for suppression of tone -- it seems to be distributed to several areas in the brainstem. Many of the cells responsible are probably cholinergic, since loss of muscle tone can be caused by injection of cholinergic agonists into the pons. The nucleus reticularis pontis oralis appears to be one of the major players since lesioning this area results in REM sleep without atonia (in humans this can be seen as REM behavior disorder).

I'd have to look up more details to be more precise.

Thanks very much, yes that sounds like the a very good summary of best model as far as my limited reading over the last two days shows. I'm very interested indeed, as it's a fairly common experience and I suspect solving it once and for all will lead us forward. I'm planning to review the medical literature over the next few days, and then I'll post a quick summary.

Again - i don't think it's any kind of woo - I'm pretty certain it has a sound neurological basis - I just don't think we should stop searching for that quite yet!

cj x
 
Obviously I don't think sleep paralysis is aliens, ghosts or witches! I'm just saying that honestly we don't acualy really understand it yet.

cj x

Hypnopompic hallucinations usually go hand-in-hand with sleep paralysis. Otherwise, we probably wouldn't even notice it.
 

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