Last night I did some research and reading somewhat randomly. I started off on quantum physics and ended on the immune system. What struck me was the very delicate (yet quite robust in terms of self-correction) balance of cellular signalling. Very small differences in voltages that cause changes. How an organism grows and how the cells communicate with each other so that they differentiate.
I had just seen this article which shows what can happen is something goes wrong.
https://www.foxnews.com/health/ct-scan-man-back-pain-3-kidneys-case-report
And yet the Telco industry people say that the only effect is heating. Well, even heating on a microscale could cause problems. But it is the ion channels (particularly the calcium channel) that can cause excessive reactive oxygen species that upset things.
And yet not one person here will concede, even with all the studies showing harm, that electrosmog may be CONTRIBUTING to the Covid-19 pandemic through a weakening or an interference of our immune system.
I was reading how tuberculosis hijacks the macrophage cell and instead of being killed by the cell it lives inside and reproduces. And I see that 25% of the global population are infected with a non-active TB. As I read this out to my wife she reads me the Facebook page she has just opened of a Canadian friend who beat one disease only to be diagnosed with TB. Coincidence strikes once more.
https://en.wikipedia.org/wiki/Memory
The time lapse between trigger hair stimulations suggests that the plant can remember an initial stimulus long enough for a second stimulus to initiate trap closure. This memory isn't encoded in a brain, as plants lack this specialized organ. Rather, information is stored in the form of cytoplasmic calcium levels. The first trigger causes a subthreshold cytoplasmic calcium influx. This initial trigger isn't enough to activate trap closure, and so a subsequent stimulus allows for a secondary influx of calcium. The latter calcium rise superimposes on the initial one, creating an action potential that passes threshold, resulting in trap closure. Researchers, to prove that an electrical threshold must be met to stimulate trap closure, excited a single trigger hair with a constant mechanical stimulus using Ag/AgCl electrodes. The trap closed after only a few seconds. This experiment gave evidence to demonstrate that the electrical threshold, not necessarily the number of trigger hair stimulations, was the contributing factor in Venus Fly Trap memory. It has been shown that trap closure can be blocked using uncouplers and inhibitors of voltage-gated channels. After trap closure, these electrical signals stimulate glandular production of jasmonic acid and hydrolases, allowing for digestion of the prey.
https://en.wikipedia.org/wiki/Antibody
Antibody and antigen interact by spatial complementarity (lock and key). The molecular forces involved in the Fab-epitope interaction are weak and non-specific – for example electrostatic forces, hydrogen bonds, hydrophobic interactions, and van der Waals forces. This means binding between antibody and antigen is reversible, and the antibody's affinity towards an antigen is relative rather than absolute. Relatively weak binding also means it is possible for an antibody to cross-react with different antigens of different relative affinities.
https://en.wikipedia.org/wiki/Magnetoreception
These organs detect very small fluctuations in the potential difference between the pore and the base of the electroreceptor sack. An increase in potential results in a decrease in the rate of nerve activity, and a decrease in potential results in an increase in the rate of nerve activity. This is analogous to the behavior of a current carrying conductor; with a fixed channel resistance, an increase in potential would decrease the amount of current detected, and vice versa. These receptors are located along the mouth and nose of sharks and stingrays.
https://en.wikipedia.org/wiki/Cytokine
Several inflammatory cytokines are induced by oxidative stress. The fact that cytokines themselves trigger the release of other cytokines and also lead to increased oxidative stress makes them important in chronic inflammation, as well as other immunoresponses, such as fever and acute phase proteins of the liver (IL-1,6,12, IFN-a). Cytokines also play a role in anti-inflammatory pathways and are a possible therapeutic treatment for pathological pain from inflammation or peripheral nerve injury. There are both pro-inflammatory and anti-inflammatory cytokines that regulate this pathway.
