Can you check this site with Biochemistry Base?

Kumar

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Oct 13, 2003
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Hello all(esp. Rolfe,TT..),

While doing my daily excercise, I got following link, which mentions somewhat biochemistry base of many conditions. It also give many details about biochemicals, bit differently & dynamically. So I got interested in it.

Can you check it for validity or some validity of mentionings on this site?

Some mentionings are:-

Anemia: Low Vitamin A, low Vit B1, low Vit B6, low Vit B12, low folate, low Vit C, low copper, low iron, low manganese, low phosphorus, high Vit B2, high Vit B15, high Vit E, high zinc, high calcium, high magnesium, low hydrochloric acid, heavy metal toxicity.

Constipation: Low magnesium, low sulfur, high calcium, high iron, low Vitamin C, high folate, low water intake, low fiber intake, heavy metal toxicity, [opt. psyllium husks].

Diabetes Type II: Low Vitamin C, low nickel, high selenium, high sodium, high/low manganese, low chromium, low sulfur, low potassium, high intake of glucosamine sulfate, [opt: EPA, stevia].

Hypertension (high blood pressure): Low magnesium, low potassium, low zinc, high/low calcium, high/low sodium, high/low phosphorus, high iron, high/low Vitamin E, high/low choline, high Vit B1, low Vit B2, low Vit B5, low Vit B15, low folate, low Co Q10, heavy metal toxicity. [opt. hawthorn, stevia].
http://www.acu-cell.com/dis.html

Stomach acid levels heavily interact synergistically with iron and manganese, whereby the absorption of both minerals is enhanced by higher HCl acid levels, and likewise, an increase in iron or manganese will generally - but not always - result in raised stomach acid levels. Since calcium and magnesium have the exact opposite effect on stomach acid levels, their interaction with iron and manganese have a major impact on medical conditions that are associated with raised or lowered stomach acid levels
http://www.acu-cell.com/femn.html



Nutritional relationships or risk factors with Cancer:

Copper: high levels (most cancers) - due to copper being an important co-factor for angiogenesis (new blood vessel formation in tumors)
low levels (colon cancer)

Zinc: high levels (left ovarian, left testicular, colon cancer)

low levels (esophageal, breast, cervical cancer)

Potassium: high levels (right ovarian, right testicular, bladder cancer)

Manganese: high levels (liver, breast cancer) [estrogen receptor-positive cancers]

low levels (right breast, stomach cancer) [estrogen receptor-negative cancers]

Iron (ferritin): high levels (liver, breast cancer)

low levels (left breast, stomach, esophageal cancer)

Protein/

Phosphorus: high levels (prostate, uterine cancer)

low levels (bone, kidney, pancreatic, lymph cancer)

Molybdenum: low levels (esophageal, stomach, * breast cancer)

Vanadiumlow levels (* breast cancer)

Selenium: low levels (lung, skin, prostate, liver, colorectal, breast cancer)

Calcium: high levels (breast, stomach, lung, prostate, pancreatic cancer) [inhibits Vit D]

low levels (colon, left ovarian, left testicular, prostate cancer) [high Zn/Ca ratio]

Magnesium: high levels (breast, stomach, pancreatic cancer)

low levels (right ovarian, right testicular, * thymus gland cancer)

Chromium:low levels (right ovarian, right testicular, * thymus gland cancer)

Sodium:low levels (pancreatic cancer)

high levels (* stomach cancer)

Germanium: low levels (* lung, liver, gastric, colon, brain cancer, sarcomas, lymphomas,
leukemia)

Chlorine:high levels (bladder, * colorectal, breast, esophageal cancer)

Fluorine: high levels (* bone, liver, colorectal cancer)

Iodine: low levels (breast cancer)

http://www.acu-cell.com/dis-can.html

I hope you will contribute for the good.

Best wishes.
 
Transcellular shifts under acidosis and alkalosis
For completeness also the changes by electrolyte shifts under acidosis and alkalosis must be mentioned. Under acidosis hydrogen ions enter the cell and potassium and free magnesium leave the cell. The hyperkalemia achieved will stimulate aldosterone secretion, which will increase renal excretion of potassium and magnesium. Lactate acidosis under ischemia or ketoacidosis under diabetes will potentiate cellular potassium magnesium efflux and aldosterone-induced renal excretion. It is very important to restore the electrolyte imbalance by potassium and magnesium supply, if the underlying cause of acidosis cannot be removed.

Under alkalosis hydrogen ions leave the cell and in compensation to this potassium and magnesium ions enter into the cell: hypokalemia will exist. If alkalosis is corrected causally, a potassium increase will take place, while for the correction of an acidosis a supply of potassium and magnesium will be necessary for recovery of electrolyte homeostasis.
http://www.mgwater.com/schroll.shtml

Can you tell Transcellular shifts under acidosis and alkalosis, of other minerals?

Whether there is so much importance of magnesium for the electrolyte homeostasis?
 

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